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We'd like to understand how you use our websites in order to improve them. Register your interest. Three new Uruguayan paracoccidioidomycosis human cases are presented. Two of them correspond to the disseminated form with metastatic lesions at the oral mucosa; the remaining one, with important pulmonary bilateral lesions corresponds to the pure chronic pulmonary form of the disease which is recognized by the first time in the country.
No doubt, these clinical forms will be found much more frequently in the endemic areas of Latin America if serological surveys are developed in the near future. This is a preview of subscription content, log in to check access.
Rent this article via DeepDyve. Borrero, J. Blastomicosis sudamericana de forma pulmonar pura. Google Scholar. Anales de la Facultad de Medicina, Montevideo 44, 5—6: — Susceptibility of hamsters and mice to Paracoccidioides brasiliensis using different routes of inoculation. Skin Tests with Paracoccidioidin and their importance. Proceeding of the first Pan American Symposium. Scientific Publication — Inmunoelectroosmophoresis-Inmunodiffusion in paracoccidioidomycosis. Sabouraudia 39— Mycopathologia vol.
Furcolow, M. Serologic Evidence of Histoplasmosis in Sanatoriums in the U. Londero, A. Paracoccidioidomicose: Classificacao das formas clinicas. Small forms and hyphae of Paracoccidioides brasiliensis in human tissue. Mackinnon, J. Anales de la Fac. Severo, L. Tesis de doctorado. Porto Alegre. The primary pulmonary lymph node complex in paracoccidioidomycosis. Smith, C. Improved culture method for the isolation of Histoplasma capsulatum and Blastomyces dermatitidis from contaminated specimens.
American Journal Clin. Witkind, J. Rectitis y lesiones perianales en la blastomicosis sudamericana experimental. Separata de G. Anticuerpos precipitantes especificos de la blastomicosis sudamericana revelados por inmunoelectroforesis. Sao Paulo. Download references. Reprints and Permissions. Asconeguy, F. Paracoccidioidomicosis: A proposito de 3 nuevos casos nacionales.
Mycopathologia 78, — Download citation. Issue Date : January Search SpringerLink Search. Abstract Three new Uruguayan paracoccidioidomycosis human cases are presented. Immediate online access to all issues from Subscription will auto renew annually.
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Perianal and/or rectal lesions in South American blastomycosis
We'd like to understand how you use our websites in order to improve them. Register your interest. They relate a case of a patient presenting a perineal lesion two years after the excision of a tumor caused by P. They comment on Mackinnon's pathogenic theory of South American Blastomycosis that presumes that lungs are the portal of entry of P. Thus, mucocutaneous lesions are secondary to a primary pulmonary localization by hematogenous route. This is a preview of subscription content, log in to check access. Rent this article via DeepDyve.
Experimental Paracoccidioidomycosis in Mice
A study was undertaken to determine the possibility of inducing paracoccidioidomycosis in mice exposed to the mycelial phase of P. Two strains of the fungus, designated FG and LA, were grown separately in media favoring the production of spores by the mycelial phase. These were collected and used for nasal instillation of cortisone treated-mice. Animals were observed for a period of 16 weeks and sacrificed periodically. The infection was not lethal. One of the strains LA proved to be more virulent than the other inducing a more severe type of pulmonary infection.
[Paracoccidioidomycosis: apropos of 3 new Uruguayan cases].
Virulence and infectivity of nine strains of Paracoccidioides brasiliensis were investigated in groups of mice which were inoculated intranasally or intravenously, and some of each were treated with corticosteroids. Fatal infections were not often seen among untreated mice, but mortality usually occurred when corticosteroids were given, regardless of the route of fungus inoculation. Prior treatment did not uniformly increase the incidence of infection, however; only in the case of intranasally inoculated mice was this effect seen. Most strains appeared to be more virulent when administered intravenously, with the exception of a single strain which, under the influence of corticosteroids, repeatedly displayed greatest virulence when given intranasally. All animals that died early in the course of the disease, irrespective of route of inoculation, always had acute pulmonary lesions and usually no other organ was involved. Animals which died later or were sacrificed always had chronic lung lesions.